Exfoliative Cytology of the Oral Mucosa in Smokers and Nonsmokers PDF Download
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Author: Alicia Susana Rubinstein
Publisher:
ISBN:
Category :
Languages : en
Pages : 194
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Book Description
Author: Alicia Susana Rubinstein
Publisher:
ISBN:
Category :
Languages : en
Pages : 194
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Book Description
Author: Clarence Towns
Publisher:
ISBN:
Category :
Languages : en
Pages : 292
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Book Description
Author: Sanaa Fouad Aboul-azm
Publisher:
ISBN:
Category : Oral mucosa
Languages : en
Pages : 340
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Book Description
Author: George Jay Wrubel
Publisher:
ISBN:
Category :
Languages : en
Pages : 32
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Author: Hussain Gadelkarim Ahmed
Publisher: LAP Lambert Academic Publishing
ISBN: 9783846501474
Category :
Languages : en
Pages : 116
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Book Description
Oral cancer is one of the major health problems in the Sudan, due to the habit of use of Toombak and cigarette smoking.Epithelial atypia (ET)was ascertained in 29 subjects and could not be ascertained in the remaining 271. Among the 29 subjects with ET, there were 11 (38%) toombak dippers, 14 (48%) cigarette smokers and 4 (14%) non-tobacco users. Among the 271 subjects without ET, there were 89 (33%) toombak dippers, 86 (32%) cigarette smokers and 96 (35%) non-tobacco users. ET was found in all the 5 control cases with OSCC. For the ET among toombak dippers and cigarette smokers, adjusted OR and the 95% CI were found to be 3 (0.91-9.7) and 4 (1.2-12.3), respectively.Exfoliative cytology is the most suitable method for implementation of oral screening program as it is both non-invasive and cheap.
Author: Suan Phaik Khoo
Publisher:
ISBN:
Category :
Languages : en
Pages :
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Book Description
A study was carried out to investigate whether smoking had any effect on the Langerhan cells in the oral mucosa, which might throw light on to the mechanism of malignant transformation of some keratotic lesions in the oral cavity. Smokers were associated with a significant reduction in the Langerhan cell population compared to non-smokers. The mean values of Langerhan cell density in light smokers and heavy smokers were 18.64/mm2 and 33.421mm2 respectively compared to 66.51/mm2 in non-smokers. There was a dose-response relation between the number of cigarette smoked daily and the effect on cell counts. These findings of a local immunological effect of smoking on oral epithelium may explain the means by which cigarette smoking contributes to the development of oral cancer.
Author: Paul Winston Montgomery
Publisher:
ISBN:
Category : Cancer
Languages : en
Pages : 264
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Author: Carl J. Witkop
Publisher:
ISBN:
Category : Keratinization
Languages : en
Pages : 280
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Book Description
Author: Galal Ahmed Saeed Omar
Publisher:
ISBN:
Category :
Languages : en
Pages :
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Book Description
Tobacco smoke cause cellular proliferation and chronic obstructive pulmonary disease (COPD), but changes in buccal mucosa cells in smokers and COPD patients remain unknown. Few studies have been done in humans to determine how smoking affects the epithelial cells of the pulmonary airways which was not thoroughly studied in previous publications that concentrated more on the buccal mucosal cells. We aim to assess the effect of smoking on cellular proliferative activity of buccal mucosa in healthy smokers and in smoking COPD patients as well as to detect early cellular proliferative activity in buccal mucosa. Our hypothesis is that tobacco smokes induce changes in buccal mucosa cells. 879 male subjects; non-smokers (controls), smokers and COPD patients were studied. Cytogenic damage was defined using the argyrophilic nucleolar organiser regions (AgNOR). Buccal mucosa cells we obtained from swabs brushing and respiratory symptoms were assessed by St. George's questionnaire. Lung function tests were measured by Vitalograph spirometer. AgNORs count, nuclear area, nuclear volume, percent cells with 5 or more AgNORs, were significantly higher in smokers than non-smokers (5.20 vs 3.27, 74.66 vs 55.65, 462.67 vs 304.01, 73.93% vs 14.75%, respectively), COPD patients, the values were 6.77, 110.42, 813.26, 96.97%, respectively. Respiratory symptoms were higher in smokers and COPD patients than non- smokers. There was significant relationship between respiratory symptoms and AgNORs count (PS 0.001). There was positive association between cigarette smoking and enhance cellular activity in oral mucosa. Packs year of smoking is significantly associated with cellular abnormality in smokers and COPD patients. Strong correlations were found between AgNORs parameters and respiratory symptoms, pulmonary function tests and pack year. Oxidative stress induced by smoking could explain our results. Smoking induces changes in nucleoprotein of buccal mucosa cells. Our hypothesis is that AgNOR expression can be used as histological biomarker in smokers to predict who is at risk of developing COPD.
Author: Gonghuan Yang
Publisher: Springer
ISBN: 9811083150
Category : Medical
Languages : en
Pages : 347
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Book Description
This book comprehensively covers the science and policy issues relevant to one of the major public health issues in China. It pulls together the prevalence pattern of tobacco use in different population and burden of the myriad of tobacco-related diseases. The book pays more attention to review the successes and failures of tobacco control policies in China, including the protect peoples from second-hand smoke, comprehensive banning tobacco advertisement promotion and sponsor, regulation of the contents of tobacco products and low tar cigarettes, warn about the dangers of tobacco, support for smokers to quit, and increasing tobacco taxation and price, as well as monitor and assessment on tobacco use and implement of prevention policy under the international background of tobacco control. The book analyse and explain the influence factors, especially interference from tobacco industry with public management theory frame for promoting tobacco control policies and looks at lessons learnt to help set health policy for reducing the burden of tobacco-related diseases. It is a helpful reference for experts in public health and epidemiologists in tobacco control, advocators and policy maker.
