The Effects of Continuous and High-intensity Interval Exercise on Postexercise Hypotension PDF Download
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Author: Cynthia Furmanek
Publisher:
ISBN:
Category : Exercise
Languages : en
Pages : 100
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Author: Cynthia Furmanek
Publisher:
ISBN:
Category : Exercise
Languages : en
Pages : 100
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Author: Brianna Roberts
Publisher:
ISBN:
Category : Exercise
Languages : en
Pages : 78
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High intensity interval training has become popular to produce various physiological benefits. There is much controversy over what type of interval training has the greatest effects. Purpose: This study was designed to compare high intensity interval groups to examine levels of post-exercise hypotension (PEH). Methods: Young, healthy volunteers (N=55) performed 24 workout sessions on a stationary bike over an 8 week period. Subjects were randomly placed into three different exercise groups: Tabata, Meyer, and steady state. Tabata (N=21) performed 20 seconds at 170% power output (PO) of VO2 max and 10 second rest for 8 bouts. Meyer (N=15) performed 30 seconds at 100% PO of VO2 max, with 60 seconds active recovery, for a total of 20 minutes. Steady state (N=19) performed 20 minutes of exercise at 90% ventilatory threshold (VT). Blood pressures were measured once a week and multiple times during those sessions. Measurements of PEH were taken 30 minutes after exercise. Results: There were significant differences in systolic blood pressure (SBP) between pre- and post-exercise but no significant differences between the three exercise groups. Averaged over eight weeks, steady state had PEH of 9.1 + 2.17, Meyer showed PEH of 8.3 + 1.83 while Tabata had PEH of 9.1 + 1.55. There was no significant difference in PEH between the weeks. Conclusions: Through this study, we have concluded that PEH occurs in SBP 30 minutes after exercise, regardless of exercise intensity. During the eight week training, PEH remained constant. Previous findings have concluded similar results, including studies involving blood pressure medication.
Author: Paulo Farinatti
Publisher: Frontiers Media SA
ISBN: 2889760774
Category : Science
Languages : en
Pages : 99
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Book Description
Disclosure statement: Topic Editor Linda Pescatello is a scientific consultant for Aytu BioScience. All other Topic Editors declare no competing interests with regards to the Research Topic subject.
Author: Danielle Kammermeier
Publisher:
ISBN:
Category : Exercise
Languages : en
Pages : 86
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Author: Benjamin C. Skutnik
Publisher:
ISBN:
Category :
Languages : en
Pages :
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Subjects with prehypertension are at risk for developing hypertension (HTN). Hypertension is associated with low-grade systemic inflammation (LGSI). Aerobic exercise training (ET) is a proven means to reduce both blood pressure and LGSI in healthy and diseased subjects. Recently, high intensity interval training (HIIT) has been show to elicit similar cardiovascular and metabolic adaptations as ET in healthy and at-risk populations in a more time efficient manner. Therefore, we hypothesized that HIIT would elicit greater reductions in blood pressure and LGSI than ET. Twelve pre-hypertensive subjects (systolic blood pressure 127.0 ± 8.5 mmHg; diastolic blood pressure 86.2 ± 4.1 mmHg) were randomly assigned to an ET group (n=5) and a HIIT group (n=7). All subjects performed an incremental test to exhaustion (VO2max) on a cycle ergometer prior to, after 4 weeks, and after 8 weeks of training. Resting heart rate and blood pressure were measured prior to and three times a week during training. LGSI was measured via high-sensitivity C-reactive protein (hs-CRP) prior to, after 4 weeks and after 8 weeks of training. ET subjects performed an eight week exercise training program at 40% VO2 reserve determined from the VOVO2[subscript]max test, while HIIT subjects performed exercise at 60% peak power determined from the VO2[subscript]max test. ET group trained four days/week while HIIT trained three days/week. ET exercised for 30 minutes continuously at a constant workload and cadence of 60 rpm while HIIT performed a protocol on a 1:1 work-to-rest ratio at a constant workload and cadence of 100 rpm. Both groups showed similar (p0.05) decreases in mean arterial (ET = -7.3%, HIIT = -4.5%), systolic (ET = -6.6%, HIIT = -8.8%), and diastolic (ET= -9.7, HIIT= -8.2%) blood pressure. HIIT decreased in LGSI ( -33.7%) while ET did not change LGSI (p0.05). VOVO2[subscript]max increased ~25% with both HIIT and ET with no differences (p>0.05) between groups. These data suggest both HIIT and ET similarly decreased resting blood pressure and increased VO2max while HIIT was effective in decreasing LGSI in subjects who were pre-hypertensive.
Author: Lynn Anne Wilson
Publisher:
ISBN:
Category : Exercise
Languages : en
Pages : 46
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Author: Michael Makela
Publisher:
ISBN:
Category :
Languages : en
Pages :
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Cardiometabolic diseases such as diabetes and cardiovascular disease are a growing threat to the quality of life of the population. Exercise is a frontline approach to treat and prevent cardiometabolic disease and its associated risk factors. The majority of individuals, however, are physically inactive and fail to meet weekly physical activity guidelines primarily due to time-constraints. High-intensity interval training (HIIT) is a time-efficient method of exercise for improving physical fitness and reducing cardiometabolic risk factors compared to moderate-intensity continuous training (MICT). Recent research indicates that the number of repetitions and duration of high-intensity intervals can be reduced without attenuation of health benefits. This study recruited nine physically inactive but otherwise healthy participants (6 female, 3 male) which engaged in a 6 week reduced-exertion HIIT protocol. Participants were assessed before and after a 2 week run-in period, and again upon completion of the exercise protocol to assess the effect on predicted aerobic capacity (VO2max), resting heart rate (HR), resting blood pressure, heart rate variability (HRV), fasting blood glucose, peak power, mean power, body mass, and body fat %. Participants improved predicted VO2max F(2,16)=6.33, p=.009, peak power F(2,16)=10.84, p=.001, and mean power F(2,16)=20.87, p=.00006, but no changes were observed in body mass, body fat %, resting HR, resting blood pressure, resting HRV, and fasting blood glucose. In conclusion, a reduced-exertion HIIT protocol with minimal time-commitment improved predicted VO2max, peak power, and mean power and is a time-efficient alternative or adjunct method of exercise for eliciting health benefits in physically inactive individuals.
Author: John E. Bickers (Jr.)
Publisher:
ISBN:
Category :
Languages : en
Pages : 0
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ABSTRACT: High intensity interval training (HIIT) and moderate intensity continuous training (MICT) are two exercise protocols that received a great deal of attention in the field of exercise science due to their health and performance enhancing capabilities. Pulse wave velocity (PWV) and heart rate variability (HRV) have also gained traction as important indictors of health and fitness. The purpose of this study was to compare the effects that HIIT and MICT have on PWV and HRV. This was a repeated measures experimental design. Participants (N = 24) were aged 18 to 35, who self-reported that they complete 150 minutes of moderate intensity exercise or 75 minutes of vigorous intensity exercise per week. All participants completed a familiarization session which included a graded exercise test to determine their heart rate peak (HRpeak). The HIIT protocol consisted of 40 total minutes, including: 4x4 minutes at 90% or greater of HRpeak, alternated by 3x3 minutes of active recovery at 65%-75% of HRpeak, and a 10-minute warm-up and 5-minute cool-down at 65%-75% of HRpeak. The MICT protocol consisted of 47 total minutes at 65%-75% of HRpeak, including a 10-minute warm-up and 5-minute cool-down at the same intensity (Tjønna et al., 2008). Five separate 2x3 repeated measure ANOVAs were used for PWV, low frequency to high frequency (LF/HF) ratio, standard deviations of NN intervals (SDNN), root mean square of successive differences (rMSSD), and HRV score. There was a significant interaction effect of protocol and time on PWV (p = .037); however each protocol elicited differing directional changes that were non-significant. The frequency domain measure of HRV, the LF/HF ratio, decreased significantly during the HIIT protocol from 5 minutes post to 30 minutes post (p = .033). All time domains (SDNN, rMSSD, HRV score) of HRV experienced significant decreases from pre-exercise to 5 minutes post and significant increases from 5 minutes post to 30 minutes post during each protocol (p
Author: Linda S. Pescatello
Publisher: Springer
ISBN: 3319170767
Category : Medical
Languages : en
Pages : 347
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Book Description
As the first primer on the effects of exercise on human hypertension, Effects of Exercise on Hypertension: From Cells to Physiological Systems provides the state-of-the-art effects of exercise on the many possible mechanisms underlying essential hypertension in humans. The book contains chapters by distinguished experts on the effects of exercise on physiological systems known to be involved in hypertension development and maintenance as well as less well known aspects of hypertension such as 24-hour ambulatory blood pressure profile and oxidative stress. An emerging area, the effects of resistance exercise training on blood pressure is also covered. A unique aspect of the book is that it covers the effects of exercise mimetics on vascular cell adaptations in order to begin to elucidate some of the cellular mechanisms that may underlie blood pressure reductions with exercise training. Lastly, the book will end with a chapter on the interactive effects of genes and exercise on blood pressure. Chapters are grouped by physiological system or mechanism. The text begins with two overview chapters; one on the general effects of aerobic exercise training and the second on the general effects of resistance exercise training on blood pressure. Each chapter begins with a bulleted list of key points. Effects of Exercise on Hypertension: From Cells to Physiological Systems will be of great value to professional individuals in cardiovascular medicine, the cardiovascular sciences, allied health care professionals, and medical and graduate students in the cardiovascular sciences and medicine.
Author: Olivier Girard
Publisher: Frontiers Media SA
ISBN: 2889454061
Category :
Languages : en
Pages : 169
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Book Description
In the past, ‘traditional’ moderate-intensity continuous training (60-75% peak heart rate) was the type of physical activity most frequently recommended for both athletes and clinical populations (cf. American College of Sports Medicine guidelines). However, growing evidence indicates that high-intensity interval training (80-100% peak heart rate) could actually be associated with larger cardiorespiratory fitness and metabolic function benefits and, thereby, physical performance gains for athletes. Similarly, recent data in obese and hypertensive individuals indicate that various mechanisms – further improvement in endothelial function, reductions in sympathetic neural activity, or in arterial stiffness – might be involved in the larger cardiovascular protective effects associated with training at high exercise intensities. Concerning hypoxic training, similar trends have been observed from ‘traditional’ prolonged altitude sojourns (‘Live High Train High’ or ‘Live High Train Low’), which result in increased hemoglobin mass and blood carrying capacity. Recent innovative ‘Live Low Train High’ methods (‘Resistance Training in Hypoxia’ or ‘Repeated Sprint Training in Hypoxia’) have resulted in peripheral adaptations, such as hypertrophy or delay in muscle fatigue. Other interventions inducing peripheral hypoxia, such as vascular occlusion during endurance/resistance training or remote ischemic preconditioning (i.e. succession of ischemia/reperfusion episodes), have been proposed as methods for improving subsequent exercise performance or altitude tolerance (e.g. reduced severity of acute-mountain sickness symptoms). Postulated mechanisms behind these metabolic, neuro-humoral, hemodynamics, and systemic adaptations include stimulation of nitric oxide synthase, increase in anti-oxidant enzymes, and down-regulation of pro-inflammatory cytokines, although the amount of evidence is not yet significant enough. Improved O2 delivery/utilization conferred by hypoxic training interventions might also be effective in preventing and treating cardiovascular diseases, as well as contributing to improve exercise tolerance and health status of patients. For example, in obese subjects, combining exercise with hypoxic exposure enhances the negative energy balance, which further reduces weight and improves cardio-metabolic health. In hypertensive patients, the larger lowering of blood pressure through the endothelial nitric oxide synthase pathway and the associated compensatory vasodilation is taken to reflect the superiority of exercising in hypoxia compared to normoxia. A hypoxic stimulus, in addition to exercise at high vs. moderate intensity, has the potential to further ameliorate various aspects of the vascular function, as observed in healthy populations. This may have clinical implications for the reduction of cardiovascular risks. Key open questions are therefore of interest for patients suffering from chronic vascular or cellular hypoxia (e.g. work-rest or ischemia/reperfusion intermittent pattern; exercise intensity; hypoxic severity and exposure duration; type of hypoxia (normobaric vs. hypobaric); health risks; magnitude and maintenance of the benefits). Outside any potential beneficial effects of exercising in O2-deprived environments, there may also be long-term adverse consequences of chronic intermittent severe hypoxia. Sleep apnea syndrome, for instance, leads to oxidative stress and the production of reactive oxygen species, and ultimately systemic inflammation. Postulated pathophysiological changes associated with intermittent hypoxic exposure include alteration in baroreflex activity, increase in pulmonary arterial pressure and hematocrit, changes in heart structure and function, and an alteration in endothelial-dependent vasodilation in cerebral and muscular arteries. There is a need to explore the combination of exercising in hypoxia and association of hypertension, developmental defects, neuro-pathological and neuro-cognitive deficits, enhanced susceptibility to oxidative injury, and possibly increased myocardial and cerebral infarction in individuals sensitive to hypoxic stress. The aim of this Research Topic is to shed more light on the transcriptional, vascular, hemodynamics, neuro-humoral, and systemic consequences of training at high intensities under various hypoxic conditions.
