The Effects of High-intensity Interval Exercise Training and Continuous Submaximal Exercise Training on Critical Power PDF Download
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Author: Lynn Anne Wilson
Publisher:
ISBN:
Category : Exercise
Languages : en
Pages : 46
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Author: Lynn Anne Wilson
Publisher:
ISBN:
Category : Exercise
Languages : en
Pages : 46
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Author: Jennifer Marie Zierke
Publisher:
ISBN:
Category :
Languages : en
Pages : 198
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Author: Laursen, Paul
Publisher: Human Kinetics
ISBN: 1492552127
Category : Sports & Recreation
Languages : en
Pages : 672
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Book Description
The popularity of high-intensity interval training (HIIT), which consists primarily of repeated bursts of high-intensity exercise, continues to soar because its effectiveness and efficiency have been proven in use by both elite athletes and general fitness enthusiasts. Surprisingly, few resources have attempted to explain both the science behind the HIIT movement and its sport-specific application to athlete training. That’s why Science and Application of High-Intensity Interval Training is a must-have resource for sport coaches, strength and conditioning professionals, personal trainers, and exercise physiologists, as well as for researchers and sport scientists who study high-intensity interval training.
Author: Bryce J. Muth
Publisher:
ISBN: 9780438931954
Category :
Languages : en
Pages : 107
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Training compliance was 96% for both the MCT and HIIT groups. Both training programs resulted in significant increases in total exercise time (MCT: 727 ± 65 vs. 789 ± 66 seconds and HIIT: 659 ± 84 vs. 752 ± 77 seconds, p0.05) and maximal oxygen consumption (VO2max) (MCT: 27.31 ± 2.54 vs. 28.71 ± 2.68 ml/kg/min and HIIT: 27.04 ± 3.15 vs. 30.60 ± 2.35 ml/kg/min, p
Author: Olivier Girard
Publisher: Frontiers Media SA
ISBN: 2889454061
Category :
Languages : en
Pages : 169
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Book Description
In the past, ‘traditional’ moderate-intensity continuous training (60-75% peak heart rate) was the type of physical activity most frequently recommended for both athletes and clinical populations (cf. American College of Sports Medicine guidelines). However, growing evidence indicates that high-intensity interval training (80-100% peak heart rate) could actually be associated with larger cardiorespiratory fitness and metabolic function benefits and, thereby, physical performance gains for athletes. Similarly, recent data in obese and hypertensive individuals indicate that various mechanisms – further improvement in endothelial function, reductions in sympathetic neural activity, or in arterial stiffness – might be involved in the larger cardiovascular protective effects associated with training at high exercise intensities. Concerning hypoxic training, similar trends have been observed from ‘traditional’ prolonged altitude sojourns (‘Live High Train High’ or ‘Live High Train Low’), which result in increased hemoglobin mass and blood carrying capacity. Recent innovative ‘Live Low Train High’ methods (‘Resistance Training in Hypoxia’ or ‘Repeated Sprint Training in Hypoxia’) have resulted in peripheral adaptations, such as hypertrophy or delay in muscle fatigue. Other interventions inducing peripheral hypoxia, such as vascular occlusion during endurance/resistance training or remote ischemic preconditioning (i.e. succession of ischemia/reperfusion episodes), have been proposed as methods for improving subsequent exercise performance or altitude tolerance (e.g. reduced severity of acute-mountain sickness symptoms). Postulated mechanisms behind these metabolic, neuro-humoral, hemodynamics, and systemic adaptations include stimulation of nitric oxide synthase, increase in anti-oxidant enzymes, and down-regulation of pro-inflammatory cytokines, although the amount of evidence is not yet significant enough. Improved O2 delivery/utilization conferred by hypoxic training interventions might also be effective in preventing and treating cardiovascular diseases, as well as contributing to improve exercise tolerance and health status of patients. For example, in obese subjects, combining exercise with hypoxic exposure enhances the negative energy balance, which further reduces weight and improves cardio-metabolic health. In hypertensive patients, the larger lowering of blood pressure through the endothelial nitric oxide synthase pathway and the associated compensatory vasodilation is taken to reflect the superiority of exercising in hypoxia compared to normoxia. A hypoxic stimulus, in addition to exercise at high vs. moderate intensity, has the potential to further ameliorate various aspects of the vascular function, as observed in healthy populations. This may have clinical implications for the reduction of cardiovascular risks. Key open questions are therefore of interest for patients suffering from chronic vascular or cellular hypoxia (e.g. work-rest or ischemia/reperfusion intermittent pattern; exercise intensity; hypoxic severity and exposure duration; type of hypoxia (normobaric vs. hypobaric); health risks; magnitude and maintenance of the benefits). Outside any potential beneficial effects of exercising in O2-deprived environments, there may also be long-term adverse consequences of chronic intermittent severe hypoxia. Sleep apnea syndrome, for instance, leads to oxidative stress and the production of reactive oxygen species, and ultimately systemic inflammation. Postulated pathophysiological changes associated with intermittent hypoxic exposure include alteration in baroreflex activity, increase in pulmonary arterial pressure and hematocrit, changes in heart structure and function, and an alteration in endothelial-dependent vasodilation in cerebral and muscular arteries. There is a need to explore the combination of exercising in hypoxia and association of hypertension, developmental defects, neuro-pathological and neuro-cognitive deficits, enhanced susceptibility to oxidative injury, and possibly increased myocardial and cerebral infarction in individuals sensitive to hypoxic stress. The aim of this Research Topic is to shed more light on the transcriptional, vascular, hemodynamics, neuro-humoral, and systemic consequences of training at high intensities under various hypoxic conditions.
Author: Thomas L. Stöggl
Publisher: Frontiers Media SA
ISBN: 2889459667
Category :
Languages : en
Pages : 167
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Author: John E. Bickers (Jr.)
Publisher:
ISBN:
Category :
Languages : en
Pages : 0
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ABSTRACT: High intensity interval training (HIIT) and moderate intensity continuous training (MICT) are two exercise protocols that received a great deal of attention in the field of exercise science due to their health and performance enhancing capabilities. Pulse wave velocity (PWV) and heart rate variability (HRV) have also gained traction as important indictors of health and fitness. The purpose of this study was to compare the effects that HIIT and MICT have on PWV and HRV. This was a repeated measures experimental design. Participants (N = 24) were aged 18 to 35, who self-reported that they complete 150 minutes of moderate intensity exercise or 75 minutes of vigorous intensity exercise per week. All participants completed a familiarization session which included a graded exercise test to determine their heart rate peak (HRpeak). The HIIT protocol consisted of 40 total minutes, including: 4x4 minutes at 90% or greater of HRpeak, alternated by 3x3 minutes of active recovery at 65%-75% of HRpeak, and a 10-minute warm-up and 5-minute cool-down at 65%-75% of HRpeak. The MICT protocol consisted of 47 total minutes at 65%-75% of HRpeak, including a 10-minute warm-up and 5-minute cool-down at the same intensity (Tjønna et al., 2008). Five separate 2x3 repeated measure ANOVAs were used for PWV, low frequency to high frequency (LF/HF) ratio, standard deviations of NN intervals (SDNN), root mean square of successive differences (rMSSD), and HRV score. There was a significant interaction effect of protocol and time on PWV (p = .037); however each protocol elicited differing directional changes that were non-significant. The frequency domain measure of HRV, the LF/HF ratio, decreased significantly during the HIIT protocol from 5 minutes post to 30 minutes post (p = .033). All time domains (SDNN, rMSSD, HRV score) of HRV experienced significant decreases from pre-exercise to 5 minutes post and significant increases from 5 minutes post to 30 minutes post during each protocol (p
Author: Benjamin C. Skutnik
Publisher:
ISBN:
Category :
Languages : en
Pages :
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Subjects with prehypertension are at risk for developing hypertension (HTN). Hypertension is associated with low-grade systemic inflammation (LGSI). Aerobic exercise training (ET) is a proven means to reduce both blood pressure and LGSI in healthy and diseased subjects. Recently, high intensity interval training (HIIT) has been show to elicit similar cardiovascular and metabolic adaptations as ET in healthy and at-risk populations in a more time efficient manner. Therefore, we hypothesized that HIIT would elicit greater reductions in blood pressure and LGSI than ET. Twelve pre-hypertensive subjects (systolic blood pressure 127.0 ± 8.5 mmHg; diastolic blood pressure 86.2 ± 4.1 mmHg) were randomly assigned to an ET group (n=5) and a HIIT group (n=7). All subjects performed an incremental test to exhaustion (VO2max) on a cycle ergometer prior to, after 4 weeks, and after 8 weeks of training. Resting heart rate and blood pressure were measured prior to and three times a week during training. LGSI was measured via high-sensitivity C-reactive protein (hs-CRP) prior to, after 4 weeks and after 8 weeks of training. ET subjects performed an eight week exercise training program at 40% VO2 reserve determined from the VOVO2[subscript]max test, while HIIT subjects performed exercise at 60% peak power determined from the VO2[subscript]max test. ET group trained four days/week while HIIT trained three days/week. ET exercised for 30 minutes continuously at a constant workload and cadence of 60 rpm while HIIT performed a protocol on a 1:1 work-to-rest ratio at a constant workload and cadence of 100 rpm. Both groups showed similar (p0.05) decreases in mean arterial (ET = -7.3%, HIIT = -4.5%), systolic (ET = -6.6%, HIIT = -8.8%), and diastolic (ET= -9.7, HIIT= -8.2%) blood pressure. HIIT decreased in LGSI ( -33.7%) while ET did not change LGSI (p0.05). VOVO2[subscript]max increased ~25% with both HIIT and ET with no differences (p>0.05) between groups. These data suggest both HIIT and ET similarly decreased resting blood pressure and increased VO2max while HIIT was effective in decreasing LGSI in subjects who were pre-hypertensive.
Author: Sam Esfandiari
Publisher:
ISBN:
Category :
Languages : en
Pages :
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Author: David G. Jenkins
Publisher:
ISBN:
Category : Exercise
Languages : en
Pages :
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